Respiratory Infection and Airway Reactivity
نویسندگان
چکیده
منابع مشابه
IL-17A inhibits airway reactivity induced by respiratory syncytial virus infection during allergic airway inflammation.
BACKGROUND Viral infections are the most frequent cause of asthma exacerbations and are linked to increased airway reactivity (AR) and inflammation. Mice infected with respiratory syncytial virus (RSV) during ovalbumin (OVA)-induced allergic airway inflammation (OVA/RSV) had increased AR compared with OVA or RSV mice alone. Furthermore, interleukin 17A (IL-17A) was only increased in OVA/RSV mic...
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Patients presenting for elective anaesthesia and surgery may be suffering with, or recovering from, a recent upper respiratory tract infection (URTI). It is a frequent clinical problem as to whether to postpone surgery in such patients as they may be more likely to suffer adverse respiratory events related to administration of general anaesthesia. Using dilute ammonia vapour as a chemical stimu...
متن کاملAlteration of airway neuropeptide expression and development of airway hyperresponsiveness following respiratory syncytial virus infection.
The mechanisms by which respiratory syncytial virus (RSV) infection causes airway hyperresponsiveness (AHR) are not fully established. We hypothesized that RSV infection may alter the expression of airway sensory neuropeptides, thereby contributing to the development of altered airway function. BALB/c mice were infected with RSV followed by assessment of airway function, inflammation, and senso...
متن کاملA role for airway remodeling during respiratory syncytial virus infection
BACKGROUND Severe respiratory syncytial virus infection (RSV) during infancy has been shown to be a major risk factor for the development of subsequent wheeze. However, the reasons for this link remain unclear. The objective of this research was to determine the consequences of early exposure to RSV and allergen in the development of subsequent airway hyperreactivity (AHR) using a developmental...
متن کاملUltrafine carbon black particles enhance respiratory syncytial virus-induced airway reactivity, pulmonary inflammation, and chemokine expression.
Exposure to particulate matter (PM) may exacerbate preexisting respiratory diseases such as asthma, chronic obstructive pulmonary disease (COPD), bronchitis, and pneumonia. However, few experimental studies have addressed the effects of PM on lower respiratory tract (LRT) viral infection. Respiratory syncytial virus (RSV) is a major etiological agent for LRT infections in infants, the elderly, ...
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ژورنال
عنوان ژورنال: Medical Clinics of North America
سال: 1981
ISSN: 0025-7125
DOI: 10.1016/s0025-7125(16)31488-2